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Tagliaferro

Anthony R. Tagliaferro, Ph.D.

Professor

Director, Center for Health Enhancement
Kendall Hall, Room 518
(603) 862-1726
Email: anthonyt@cisunix.unh.edu

Educational Background:

Ph.D., Cornell University, 1978

M.S., Lehigh University, 1972

B.S., Boston College, 1968

Courses Taught:

Clinical Nutrition (NUTR 773/873); Critical Issues in Nutrition (780/880); Disorders in Energy Balance (ANSC 855)

General Area of Interest and/or Specialty:

Human obesity; dietary, metabolic, and hormonal factors; obesity, insulin resistance and disease

Description of current research and/or professional activities:

Research interests have been focused on the dietary and metabolic factors that may cause human obesity and the mechanisms by which obesity may cause development of chronic health conditions particularly breast cancer and asthma.

In collaboration with faculty in the Departments of Kinesiology, Medical Laboratory Science and a practicing physician, we have recently formed the Center for Health Enhancement (CHE) at UNH. CHE will be an academic, research, and service oriented program that will involve training in the use and practice of state of the art methodologies of health screening. Further information can be obtained from CHE's web site, www.che.unh.edu.

Representative Publications:

Tagliaferro AR, Ronan AM. Long-term effects of sex steroid depletion on insulin growth factor-1 (IGF-1), energy metabolism, and adiposity in the pig. FASEB J. 13(4):A369, 1999.

Tagliaferro AR, Ronan AM. Dehydroepiandrosterone (Dhea): a modulator of fatty acid oxidation in energy metabolism. FASEB J 12(5);A5083, 1998.

Bennet B, Ronan AM, Tagliaferro AR. Dietary fat, DHEA, and substrate utilization. Am J Clin Nutr 71(5);A75, 1997.

Tagliaferro AR, Ronan AM, Meeker LD, Thompson HJ, and Scott AL. Cyclic food restriction, insulin, and mammary cell proliferation in the rat. Carcinogenesis 18(11);2271-2276, 1997.

Tagliaferro AR, Ronan AM, Meeker LD, Thompson HJ, Sinha D. Cyclic food restriction alters substrate utilization and abolishes protection from mammary carcinogenesis in female rats. J Nutr 126;1398-1405, 1996.

Tagliaferro AR, Ronan AM, Tse S, Meeker LD. Increased lipolysis to beta adrenergic stimulation following dehydroepiandrosterone treatment in rats. Am J Physiol 268;R1374-R1380, 1995.